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Occupational Health Safety estimated in 2002 that 1117 lung cancer deaths were caused by PM2.5 exposure, a fraction of 11% . Unlike the short-term effects linked to an inflammatory response, impacts of PM on carcinogenesis come from prolonged exposure. When mucociliary and alveolar clearance functions are exceeded, PM persist into lungs, leading to thickening of bronchial walls, an airway remodeling characterized by the hyperplasia of goblet and smooth muscle cells and a subepithelial fibrosis, as has been demonstrated in asthma and COPD . Thereby, PM

 

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